SnapShot: B7/CD28 Costimulation

نویسندگان

  • Alison M. Paterson
  • Vijay K. Vanguri
  • Arlene H. Sharpe
چکیده

B7-1 (CD80) Inducible: APCs (slower kinetics than B7-2), T cells CD28 Constitutive: mouse (all T cells), human (95% of CD4+, 50% of CD8+ T cells), plasma cells, NK cells Stimulation of naive and previously activated T cells upon TCR ligation: ↑ proliferation (↓ p27Kip1, ↑ G1/S); ↑ cytokine production (↑ NFκB, ↑ IL2); ↑ survival (↑ Bcl-xL, ↑ survival of Tregs); ↑ glucose metabolism; provides T cell-dependent B cell help for class switching. Blockade of pathway to treat autoimmune infl ammatory diseases and for transplantation; active engagement to expand anti-tumor T cells and Tregs B7-2 (CD86) Constitutive at low levels; inducible: APCs, T cells

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The capacity of the natural ligands for CD28 to drive IL-4 expression in naïve and antigen-primed CD4+ and CD8+ T cells.

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The role of B7 costimulation in CD4/CD8 T cell homeostasis.

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Critical role of CD28/B7 costimulation in the development of human Th2 cytokine-producing cells.

CD28 is a major costimulatory signal receptor for T cells. We have used human naive CD4+ cells from cord blood to analyze the effect of the CD28/B7 costimulatory pathway on development of T helper (Th) subsets. We show that CD28 costimulation is critical for development of the Th2 cytokine-producing cells and that in the absence of CD28 costimulation, cells are not primed to produce Th2 cytokin...

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Expression and contribution of B7-1 (CD80) and B7-2 (CD86) in the early immune response to Leishmania major infection.

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عنوان ژورنال:
  • Cell

دوره 137  شماره 

صفحات  -

تاریخ انتشار 2009